By David C. Whitcomb MD PhD, Adam Slivka MD, Kenneth K. Lee
Problems of the pancreas have, some time past, been very complex as the analysis was once usually made past due throughout the ailment and no considerably helpful interventions have been to be had. this case is speedily altering as new insights from a number of views are built-in and serious about every one step of this advanced techniques. This factor of Gastroenterology Clinics of North the United States highlights a few parts of fast development in inflammatory and neoplastic problems of the pancreas. every one bankruptcy represents the built-in wisdom and standpoint of specialists within the box, and characterize the hottest research of those cutting-edge methods to complicated matters within the assessment and remedy of pancreatic problems.
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Extra info for Advances in the Diagnosis and Treatment of Pancreatic Diseases, An Issue of Gastroenterology Clinics Vol 36 Issue 2
The National Institutes of Health published a consensus statement in 2002 that recommends that diagnostic ERCPs should be avoided whenever possible . MECHANISMS OF POST–ENDOSCOPIC RETROGRADE CHOLANGIOPANCREATOGRAPHY PANCREATITIS There have been numerous theories about the mechanisms of post-ERCP pancreatitis. The most widely accepted theory is that mechanical trauma to the papilla or pancreatic sphincter, caused during instrumentation, creates transient obstruction of outflow of pancreatic juice.
36] Khabiri AR, Bagheri F, Assmar M, et al. Analysis of specific IgE and IgG subclass antibodies for diagnosis of Echinococcus granulosus. Parasite Immunol 2006;28(8):357–62. AUTOIMMUNE PANCREATITIS 255  Jaoko WG, Simonsen PE, Meyrowitsch DW, et al. Filarial-specific antibody response in East African bancroftian filariasis: effects of host infection, clinical disease, and filarial endemicity. Am J Trop Med Hyg 2006;75(1):97–107.  Bhunia B, Bhandary YP, Reddy MV, et al. Analysis of IgG subclasses and IgE antibodies across the clinical spectrum of bancroftian filariasis in an endemic area.
Suspected sphincter of Oddi dysfunction (SOD) has been found to increase greatly a patient’s risk for developing post-ERCP pancreatitis. 001). 05%) was markedly elevated . 09 . A prior history of post-ERCP pancreatitis also confers a considerable increased risk for recurrence of this complication. 4%. [1,2,6,7]. In addition, if a patient has a history of recurrent acute pancreatitis, the incidence of pancreatitis ranges from 16% to 23% [4,6]. Unfortunately, these risk factors for post-ERCP pancreatitis are also thought synergistically to increase a patient’s risk for postprocedure pancreatitis regardless of the type of endoscopic therapy performed.
Advances in the Diagnosis and Treatment of Pancreatic Diseases, An Issue of Gastroenterology Clinics Vol 36 Issue 2 by David C. Whitcomb MD PhD, Adam Slivka MD, Kenneth K. Lee